Male testis is an organ that produces sperm, so it is necessary to do a good job of testicular care in life. If there is something wrong with the testicles, it will affect fertility. So do male testicles get smaller and need treatment? What is the situation that male testicles become smaller? There are many reasons why male testicles become smaller. Generally, no treatment is needed. Let's take a closer look.

1, male testicles become smaller.

Some people's testicles are big and small from childhood, and the difference is very obvious, mostly caused by congenital diseases. There are two common congenital causes: one is congenital testicular dysplasia, which is caused by a disease called Coffert syndrome, which is also called seminiferous tubule hypoplasia. About one in five hundred boys. This disease is mainly caused by abnormal sex chromosomes. A normal male has 46 chromosomes, represented by 46, XY. Patients with Creutzfeldt-Jakob disease have an extra sex chromosome X with a karyotype of 47, XXY. Part of the reason is that the mother's oocytes did not undergo meiosis during mitosis to form double X eggs, and part of it is caused by the abnormal sperm production of the father.

This kind of person has no abnormality in childhood, but gradually shows abnormality in adolescence or adulthood. Most of these patients have delayed puberty, that is, after 14 years old, the testicles are still as big as children, and the secondary sexual characteristics of men are not obvious, such as tall figure, slender limbs, narrow shoulders and pelvis.

Similar to women, they are broad and muscular, but they have more subcutaneous fat and are prone to obesity. The voice is sharp and thin, and the Adam's apple, beard and body hair are not obvious or sparse. 80% patients can have female manifestations such as breast enlargement, but they don't secrete milk. The external genitalia is male. Tintin is normal or short. Although he is an adult, his testicles are small and hard. The mental development of such patients can be normal or slightly delayed. Another microtubule accretion is congenital adrenal hyperplasia, which is caused by congenital lack of 2 1- hydroxylase. This enzyme must be involved in the process of adrenal gland synthesizing cortisone from cholesterol. When 2 1- hydroxylase is deficient, cortisone synthesis will decrease. On the contrary, the synthesis of androgen (testosterone) in adrenal gland has greatly increased.

Under the action of too much androgen, the patient appears precocious puberty, and male secondary sexual characteristics appear before 10 years old, but the testicles are smaller. This is because testosterone produced by adrenal gland can inhibit the secretion of gonadotropin by pituitary gland, and then inhibit the development of testis, so this precocious puberty is also called pseudoprecocious puberty. In addition to these two common congenital reasons, there are many acquired factors that lead to different testicular sizes. For example, the trauma of one testicle can cause testicular bleeding and hematoma, resulting in insufficient blood supply to the testicle and gradual atrophy, which is obviously smaller than that of the opposite side.

Mumps virus can destroy the epithelial cells of seminiferous tubules in testis, which is also one of the important reasons for the large and small testis. In addition, some people mistake other pathological changes of scrotum for testicles of one big and one small. For example, the hydrocele in the tunica vaginalis is only the water sac around the testis, not the enlargement of the testis itself. Some people mistake indirect inguinal hernia, varicocele, epididymal tuberculosis and epididymitis for testicular enlargement, because these diseases sometimes show that one scrotum is bigger than the other.

Normal people's testicles are not equal on both sides, but the difference is not too big. If the testicles on both sides are within the normal size range, it will not affect fertility. But if one side is a small testicle and the other side is normal, although its fertility is lower than that of normal people, it can still give birth. But at the same time, it must be pointed out that, generally speaking, testicular damage is only due to the destruction of internal spermatogenic cells, but the interstitial cells that produce testosterone are not destroyed, and testosterone can still be produced, maintaining male secondary sexual characteristics and sexual function.

Therefore, the testis becomes smaller, which only affects fertility and does not affect sexual function. The above-mentioned patients with Creutzfeldt-Jakob syndrome can still have sexual desire and normal ejaculation because their testis can secrete a certain amount of androgen, but 97% of them are infertile because of testicular spermatogenesis disorder, seminiferous tubule atrophy, no sperm in semen or very few sperm.

The treatment of microtubule accretion should be carried out according to different conditions. For example, varicocele leads to different testicular sizes. As long as varicocele surgery is performed, the problem will be solved. However, the treatment of microtorchiasis caused by congenital factors, such as Creutzfeldt-Jakob syndrome, testosterone propionate 50 mg, intramuscular injection twice a week, or methyltestosterone 5 mg, three times a day, can promote the development of male secondary sexual characteristics, promote desire and maintain normal sexual function.

For those with mild symptoms and fertile testis, intramuscular injection of 2000 units of chorionic gonadotropin, every 2 days 1 time, can promote testicular development, but the effect is not ideal. No matter what treatment method is adopted, it can only promote the development of sexual function and secondary sexual characteristics, but can not restore fertility.

2. Good food for testicles

1, food containing zinc

Zinc is an indispensable trace element in human body and plays an important role in maintaining the normal structure and function of male reproductive system. Zinc deficiency will reduce the number of sperm, affect desire and reduce sexual function. Oyster meat is the food with the highest zinc content. Other foods such as beef, milk, chicken, chicken liver, egg yolk, shellfish, peanuts, cereals, beans, potatoes, vegetables and brown sugar all contain a certain amount of zinc.

2. Oats

The plant extract can improve the testosterone level by promoting the release of luteinizing hormone. Perhaps this is why oatmeal is a must-have diet for many bodybuilders. A bowl of oatmeal may not contain enough active ingredient, oat saponin, to produce enough stimulation, but concentrated oat extract can. Oat saponin is a kind of plant saponin similar to furasterol saponin.

3. Foods rich in magnesium

Magnesium helps to regulate human heart activity, lower blood pressure, prevent heart disease and improve male fertility. Foods with high magnesium content include soybeans, potatoes, walnuts, oatmeal, macaroni, leafy vegetables and seafood.