Uric acid poisoning is mainly formed in muscle tissue. In the case of high-intensity exercise or hypoxia, muscle tissue will produce a large number of uric acid acidosis through anaerobic metabolism. Uric acid poisoning can play multiple roles in the body. It can provide extra energy for muscle tissue undergoing high-intensity activities; It can also adjust the pH of blood and maintain the stability of blood during strenuous exercise.
Uric acid poisoning is closely related to training intensity and physical condition. Under normal circumstances, our body can decompose lactic acid by supplying oxygen, convert it into carbon dioxide and water, and excrete it. However, in the case of high-intensity exercise or hypoxia, lactic acid will be further metabolized into uric acid due to insufficient oxygen supply. In this case, uric acid will accumulate in muscle tissue, causing muscle soreness and fatigue.
The production and elimination of uric acid is a dynamic equilibrium process. When the intensity of exercise drops or rests, the body will gradually remove the accumulated uric acid. However, if you do continuous strenuous exercise or lack of rest, uric acid poisoning may accumulate in the body, leading to lactic acidosis.
In order to reduce the accumulation of uric acid and delay the occurrence of lactic acid fatigue, some strategies can be adopted in training. Reasonable control of training intensity and exercise time to avoid long-term high-intensity exercise. Increase the proportion of aerobic exercise and improve the body's ability to use oxygen. It is also important to supplement carbohydrates and water properly, which can provide enough energy and water to promote the metabolism and elimination of uric acid.
In addition to exercise, uric acid poisoning is also related to some diseases. For example, due to insufficient insulin secretion or cell insensitivity to insulin in diabetic patients, glucose in blood can not be effectively utilized, and a large amount of lactic acid and uric acid are produced during metabolism. This is also one of the reasons why diabetic patients are prone to hyperlactic acidosis.
Urinary acetic acid is an organic acid produced under high intensity exercise or hypoxia. It plays multiple roles in muscle tissue, but excessive accumulation will cause lactic acid fatigue. Reasonable control of exercise intensity, increasing aerobic exercise, supplementing carbohydrates and water can reduce the accumulation of uric acid. In some diseases, it is also related to the formation of uric acid. Therefore, it is necessary to pay attention to the influence and treatment of uric acid in exercise and disease treatment.