This is tinea pedis caused by ringworm infection, commonly known as athlete's foot. It is a dermatophyte infection that occurs on the skin on the soles of the feet and between the toes. Sometimes it can extend to the heels and insteps, but it only occurs Those on the dorsum of the feet are called tinea corporis.

This disease mostly occurs in middle-aged and young people, rarely in children, and is generally more common in men than women. Due to different pathogenic bacteria, patients' health status and physical differences, their clinical manifestations also vary. There are four common types:

1. Sweat blister type. The initial onset is mostly scattered thick small blisters, which rupture to form annular scales or multiple annular scales, which can gradually expand into sheets, causing the skin to gradually thicken and cause conscious itching.

2. Interdigital type. It usually occurs between the 3rd and 4th or between the 4th and 5th toes, with a small amount of scales and severe itching. Patients often scratch unconsciously, and sometimes the scales are not obvious and they only feel itching. This is called asymptomatic type, but they are often carriers of bacteria. Scratching can easily lead to secondary bacterial infection, local erosion, moisture and exudate.

3. Scaly keratosis type. It manifests as localized, unshaped, erythrophilic scaly patches on the soles, heels and lateral edges of the feet. The surface is obviously keratinized, rough and dry, and the boundaries are clear or unclear. Symptoms are more severe in winter and can easily lead to chapped skin. A few papules or vesicles often appear in summer. The course of the disease is chronic. Over time, the skin may thicken and the toenails may become easily involved. This type is mostly caused by Trichophyton rubrum. The disease is stubborn and difficult to cure.

4. Moist erosion type. This type is mostly secondary to interdigital type or sweat blister type. It is mostly caused by excessive scratching, which often leads to secondary bacterial infection. The local inflammation is obvious, soaked and eroded, there is obvious exudation, and it is itchy and painful. It is more common in summer.

Treatment methods:

1. Local drug treatment, symptomatic treatment according to different clinical manifestations of the disease:

(1) For sweat blister type or symptoms For mild erosion and a small amount of secretion, soak or wet compress with 1:6000 or 1:4000 potassium permanganate solution or 3% lead acetate solution every morning and evening for 20 to 30 minutes each time (blisters can be treated aseptically Prick the bottom), soak it and wipe it dry with a sterile cloth. Then choose the following agents for external application, such as benzoic acid/salicylic acid/iodine/potassium iodide/menthol (compound benzoic acid tincture), Castellan liniment, 5% fish oil, 3% salicylic acid, 10% sulfur paste, or commercially available Sold over-the-counter medicines, such as 2% clotrimazole cream, terbinafine cream, bifonazole cream, etc. (choose one), use irritating agents with caution.

(2) For erosion types with a lot of exudation, you can use the above-mentioned potassium permanganate solution or soak or wet compress with Daribo solution. For those with secondary bacterial infection, you can add 0.1% as appropriate. After thacridine (Rivanol) has stopped leaking and the erosion has improved, you can use the above-mentioned sweat blister mud ointment, half the amount of White's tincture or commercially available antifungal cream.

(3) For interdigital type, please refer to the treatment of sweat blister type.

(4) Due to the long course of the disease, the treatment of scaly keratosis requires patience. Start using tinctures with strong antifungal and exfoliating effects (need to have stronger penetration), such as compound hibiscus bark tincture or full amount of benzoic acid/salicylic acid/iodine/potassium iodide/menthol (compound benzoic acid tincture). Soak your feet in 10% to 30% acetic acid for 20 to 30 minutes each time. After the hyperkeratosis is significantly relieved, use a relatively weak ointment for external application, such as half the amount of compound benzoic acid ointment or 10% to 20% urea ointment, to consolidate the curative effect and restore normal skin barrier function.

(5) Fire-scorched sand is formed under the special geological conditions of hot magma acting on sandy soil. The formation temperature is more than 3,000 degrees, so it is very dry, contains no volatile components, and contains a variety of trace elements that are beneficial to the human body brought from deep magma. Fire-scorched sand can shrink the fimbriae and flagella of athlete's foot fungi, causing them to lose their activity and adsorption power. The bacteria are dehydrated and shriveled, in a 'coma' dormant state and lose their pathogenicity. Naturally shed from human tissue cells. At the same time, the beneficial trace elements contained in burnt sand can accelerate the repair of skin tissue. Fire burnt sand is easy to use, does not burn the feet, has no irritation, does not relapse, and does not damage the skin. Not bad indeed.

2. Systemic drug treatment. For those who have poor local treatment efficacy or have extensive skin lesions, short-term oral antifungal drugs such as itraconazole, fluconazole, terbinafine, etc. can be combined with the treatment, usually 1 to 2 weeks.