The cause of disease
1. Maternal injury
It is the main cause of uterine prolapse. Delivery, especially dystocia, prolonged second stage of labor or vaginal surgery, is likely to cause damage to the cervix, cervical main ligament, uterosacral ligament and pelvic floor muscles. If the postpartum supporting tissue fails to return to normal, uterine prolapse is prone to occur.
2. Increased abdominal pressure
Puerperal women prefer to lie on their backs and are prone to chronic urinary retention. The uterus tends to the posterior position, and the direction of the uterine axis is the same as that of the vaginal axis. When the abdominal pressure increases, the uterus will descend in the direction of vagina and prolapse will occur. Chronic constipation and cough, ascites or abdominal obesity can all increase abdominal pressure and promote uterine prolapse.
3. Congenital dysplasia
Puerperal uterine prolapse is caused by poor development of supporting tissues of reproductive organs.
4. Malnutrition
Severe nutritional deficiency can lead to muscle atrophy, pelvic fascia relaxation and loss of support for the uterus. Uterine prolapse caused by malnutrition is often accompanied by symptoms such as gastroptosis and abdominal wall relaxation.
ageing
The decline of ovarian function leads to the decrease of estrogen secretion, which makes the supporting tissue of pelvic floor weak and relaxed, prone to uterine prolapse, or aggravates the original prolapse.
clinical picture
The patient consciously falls down in the abdomen, especially when walking and squatting. Vaginal prolapse, a patient with mild prolapse, can come out on his own after lying down and having a rest. In severe cases, he can't come out, which affects his actions. Due to long-term exposure, the surface of cervical mucosa is thickened, keratinized, eroded and ulcerated. Patients with increased leucorrhea, sometimes purulent or bloody, some menstrual disorders, excessive menstrual blood. When accompanied by cystocele, dysuria, urinary retention and stress urinary incontinence may occur.
Uterine prolapse is the downward displacement of the uterus along the vagina, which can be divided into 3 degrees according to the degree of prolapse:
1. 1 degree
Refers to the level of the cervical external orifice is lower than the sciatic spine, not reaching the edge of hymen, and the cervix and uterus are still in the vagina. This degree of uterine prolapse does not need treatment, and it can be recovered after a rest.
2. Ⅱ degree
Refers to the cervix has slipped out of the vaginal opening, and the uterine body or part of the uterine body is still in the vagina. However, due to the large scope, the cervix only exits the vaginal opening. In severe cases, due to the elongation of the cervix, the extended cervix and vaginal wall both exit the vaginal opening.
Second degree uterine prolapse can be divided into mild and severe types: ① second degree mild? The cervix is out of the vagina, and the uterus is still in the vagina. ② Ⅱ degree weight? Most or all of the cervix, part of the uterus and the anterior vaginal wall are outside the vaginal orifice.
3. Ⅲ degree
Refers to the whole uterine body and cervix coming out of the vagina.
Pelvic floor muscle exercise: suitable for mild patients. Levator ani's exercise method is: forcibly contract the anus, and relax the pelvic floor muscles after contracting for more than 3 seconds, lasting 10 ~ 15 minutes each time, 2 ~ 3 times a day. The first exercise should be done before getting up.
Vitamin O in Phoenix Blood Maintains Four Functions of Uterus
1, MSCs homing to the damaged part of endometrium can inhibit local inflammatory injury through immunomodulation. MSCs can produce a large number of anti-inflammatory cytokines and growth factors after migrating to the damaged site.
2.MSCs can induce angiogenesis at the site of endometrial injury. After the transplanted MSCs enter the injured site, they can autocrine and paracrine to produce cytokines, such as vascular endothelial growth factor.
3. Previous studies have proved that stem cells can treat intrauterine adhesions by promoting the regeneration and repair of endometrium. It can secrete a variety of cytokines, inhibit cell apoptosis and repair damaged tissues and cells.
4. It may change the expression of some genes, and achieve the function of regulating uterine function through gene changes.